Methyl jasmonate, as a plant hormone and signal molecule related to injury, widely exists in plants and can be used in artificial cultivation of jasmonate oil.
Exogenous applications of JA (JA) and MeJA (Methyl jasmonate) can stimulate the expression of defense plant genes, induce chemical defense in plants, and produce effects similar to mechanical damage and insect feeding. A large number of studies have shown that treatment plants can system with jasmonic acid compounds induce protease inhibitor (PI) and polyphenol oxidase (PPO), thus affecting plant-eating animals to absorb nutrients, also can increase peroxidase, chitosan enzyme and the activity of lipoxygenase and other defense protein levels, phenolic acids of alkaloids and the accumulation of secondary substances, increase and change of volatile signal compounds released, even defensive structures, such as trichomes and resin ducts. Plants treated with JASmonic acid increased the death rate of herbivores and became more attractive to predatory and parasitic predators. The volatile compound, methyl jasmonate, can enter the plant from the stomata and be hydrolyzed to jasmonic acid in the cytoplasm by esterase, realizing long-distance signal transduction and interplant communication, and inducing the adjacent plants to produce the induced defense reaction. Jasmonic acid and methyl jasmonate have four kinds of stereoisomerism respectively, among which the active cis-type structure is cis-type structure, but the cis-type structure is unstable and will be different from the isomerization to the trans-type structure. The metabolite of jasmonic acid (Z) -CIS-Jasmone (CIS-JASmone) is electrophysiologically active and plays a role in plant-induced defense, and it is different from jasmonic acid and methyl jasmonate in the role of defense signals. One of the aroma components of black tea. It has the sweet smell of jasmine. . Jasmonate accounted for 1% of the peak area in the aroma group in the seed – packed tea, but only trace amount in jasmine tea.